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Previous studies have shown that H 2 S donors or related release systems, such as S-propargyl-cysteine (SPRC), can suppress the expression of M1-related inflammatory factors and promote the formation of M2-like phenotypes, suggesting that the Cys-GSH/H 2 S axis may jointly restrain pro-inflammatory programs by lowering the oxidative stress threshold and modulating signal transduction (79)
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What the research suggests and where it falls short This is the section that matters most with DSIP, because the honest answer is uncomfortable: the evidence is genuinely weak and conflicting
Lipid hydroperoxides, generated primarily through the activity of lipoxygenases (LOXs), are key mediators of cellular dysfunction and death ( 2.1.3 Antioxidant system 2.1.3.1 System Xc-/GSH/GPX4 axis The cystine/glutamate antiporter (System Xc-), glutathione (GSH), and glutathione peroxidase 4 (GPX4) form a critical antioxidant axis that plays a central role in regulating ferroptosis
This means it primarily targets GH release without significantly impacting other hormone levels, such as cortisol, acetylcholine, prolactin, and aldosterone
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