In Alzheimers disease, GLP-1 RAs have been shown to improve glucose transport across the blood-brain barrier and reduce inflammation in neural pathways
After reconstitution with bacteriostatic water, store at 2-8C and use within 28 days
It may increase glucose uptake by breaking down TAGs in chylomicrons through LPL activity and transporting glucose through a protein called GLUT4
TEN significantly reduced glial fibrillary acidic protein immunoreactivity and astrocyte activation, implying that its analgesic effects are associated with suppression of spinal astrocytes and neuroinflammation [36, 37]
Benichou O, Coskun T, Gonciarz MD, Garhyan P, Adams AC, Du Y, et al
Research in neurodegenerative diseases like Alzheimers suggests these medications may reduce neuroinflammation and protect against beta-amyloid plaque formation