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In the context of wound healing, oxidative stress can lead to the existence of a prolonged pro-inflammatory environment as well as dysregulated re-epithelialisation, as discussed in the following sections 58

The Journal of biological chemistry, 289(28), 1950819518

In particular, in studies where only the 5 mM concentration of LC or ALC reduced infarct size significantly, prolonged drug administration could be a factor influencing the effectiveness of protection

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