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Our present observations are in keeping with our previous findings (17,30,31) indicating that the production of free radical, enhanced lipid peroxidation and an impairment of antioxidizing enzyme activity, and the increased generation of cytokines, such as IL-1 and TNFalpha, contribute to the pathomechanisms of stress-induced gastric lesions

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To view or add a comment, sign in An interesting prespecified analysis from the FOURIER trial, highlighting how obesity is not only a cardiovascular risk factor but also a modifier of therapeutic benefit

this makes it a potential candidate for treating alcoholinduced endotoxemia