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Acknowledgments The authors wish to acknowledge the patients who consented to be included in the current case series
The active transport of Ca 2+ is mainly regulated by the biologically active form of vitamin D, 1,25(OH) 2 D 3 (calcitriol)[8], by previous activation of a vitamin D receptor (VDR)[9]
Iron-associated lipid peroxidation in Alzheimers disease is increased in lipid rafts with decreased ferroptosis suppressors, tested by chelation in mice
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The effects of CR on younger individuals are controversial
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