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With respect to glycolysis in obese adipocytes, a decrease in glucose 6-phosphate accompanied by increased intracellular glucose levels was observed, which implied hexokinase as a possible major rate-limiting step in the provision of glucose carbons required for TG synthesis [32]

Therapeutic GHK-Cu simply restores levels that have declined with age bringing them closer to what you had at 20 rather than introducing something foreign

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Several novel GIP analogues have been developed which act as stronger GIP agonists, showing resistance to degradation by Dipeptidyl Peptidase-4 (DIPP-4) [135] and demonstrating increased insulinotropic and blood glucose lowering activity [135]