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This suggests the GcgR-G s -cAMP axis to be responsible for generating maximal glycogenolytic and gluconeogenic effects under pharmacological treatment, and thus is a likely mediating pathway of glucagons acute enhancement in energy expenditure [102], [152], [188]

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Through molecular interaction experiments (such as Co-IP and dual-luciferase reporter gene assays), the regulatory mechanisms between p53 and key target molecules such as SLC7A11 and FOXO3 can be analyzed to elucidate the molecular switch of its bidirectional effects

Younis, S., Hougaard, A., Noseda, R