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Because of spatial constraints, genes must engage in a complex unfolding process to become accessible to the transcriptional machinery, which is triggered through posttranslational modifications at the N-terminal tails of core histones

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However, in another study enrolling 172 STEMI-patients using as endpoint CMR salvage index derived from myocardial area at risk in the acute phase, and infarct size by LGE at follow-up (90 21 days after pPCI) exenatide treatment resulted in a significantly larger salvage index and a smaller infarct size (when related to the myocardial area at risk), despite no differences in LVEF or significant changes in the absolute infarct size [147]

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