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Discussion Pathophysiology of CMD CMD involve interplay of a cascade of pathophysiological events ensuing in a rise in IR, accumulation of free fatty acids (FFA) in the circulation, lipid and glucose dysmetabolism, and raised levels of adipokines and cytokines [19,20,21]

2,6 Although the majority of patients experience mild adverse reactions, such as hepatitis, cholestasis or asymptomatic liver enzyme elevation, APAP hepatotoxicity is generally estimated to account for approximately 48% of acute liver failure diagnoses

In recent computational studies, AG1 was used as a scaffold for a structure-based pharmacophore screen where five compounds were identified as potential activators of G6PD [102]

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