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Cells were stimulated by 20 M GLP-2 together with the camelid-derived nanobody Nb35 to stabilize the interface between G s and G
The aim of this study is to exploit GSH-responsive AP site capture reagent (AP probe-net), which responses to the elevated glutathione (GSH) levels in the tumor micro-environment (TME), releasing reactive alkoxyamine to trap AP sites and block the APE1-mediated BER for targeted anti-tumor activity against ATC
