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Thymosin Beta-4 is typically examined through systemic research protocols to observe its role in cellular motility

Discussion Despite the development of a comprehensive treatment model incorporating surgery, chemotherapy, and immunotherapy, the prognosis of GC patients remains unfavorable [41]

In addition to the GPX4-dependent mechanisms, several GPX4-independent pathways contribute to ferroptosis regulation, including the NAD(P)H/FSP1/CoQ10 axis, the GCH1/BH4/DHFR axis, squalene accumulation, and DHODH-mediated suppression of ferroptosis [88, 92]

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