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2.3 Aberrant changes in mitochondrial structure and function during AKI 2.3.1 Mitochondrial dysfunction Mitochondrial dysfunction in AKI is characterized by impairments in mitochondrial biogenesis, attenuation of mitophagy, disrupted mitochondrial dynamics, and reduced OXPHO activity (Figure 1) (Zhu et al., 2023
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In murine hepatocytes, excess administration of iron promotes ferroptosis 16
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43 The relevance of this pathway for tumorigenesis is demonstrated by the fact that humans with the Laron syndrome, a recessively inherited defect in the growth hormone receptor, display extremely low IGF-1 and reduced insulin concentrations and usually do not develop cancer despite high prevalence of obesity and dyslipidemia
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