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This signalling pathway, previously described in the regulation of immune responses in murine macrophage models, is essential for macrophage differentiation toward the M2 phenotype [53,54]

The relationship is tripartite: Selenium Selenocysteine Glutathione Peroxidase (GPx) HO Neutralization Requires Glutathione (GSH) as substrate Selenium provides the enzymatic machinery

doi: 10.1038/355446a0

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Upregulated gene sets included those associated with GPCR signaling, neuropeptide receptor interactions and ligand binding, cilia formation, metabolic processes, and endocrine function Supplemental Figure 5B)