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Additionally, hypoxia-induced downregulation of nuclear receptor coactivator 4 and p53 polymorphisms also control the susceptibility of macrophages to ferroptosis (50, 51, 84)

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Since cysteine is known to be neurotoxic in excessive amounts [32], treatment with N-acetylcysteine should not be recommended for patients with glutathione synthetase deficiency as this may increase the intracellular cysteine levels even more

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